Protocols v49




Protocols — RemedyLink


Protocols

Welcome. These protocols are arranged in order, and many depend on the ones before them being in place. Work through them in sequence; the final protocol, the spike protein protocol, is added only when symptoms persist and the clinical picture calls for it. For the science behind these protocols, see Notes of a Master Formulator. To view the charts that accompany the Protocols, go to Remedylink.com/charts.










Stool Test

The cheapest, most informative diagnostic in functional medicine

Six pass/fail tests

1. Stool pH between 5.7 and 6.2

6.0-8.0 pH paper strips. Press against stool on TP, or defecate on newspaper. Read the strip at 15 seconds, and take the most alkaline reading. Do not test stool that has gone into toilet water. Blue = alkaline (trouble). Green = neutral (not terrible, not great). Yellow-green / unchanged = acidic (success).

2. No toilet paper needed

A fully formed stool with adequate fiber and normal mucus leaves nothing behind. Toilet paper is like tissue paper. You only need it if you’re sick. Extensive wiping = upstream issue.

3. Sinking stool — minimal gas

Healthy stool sinks. Dysbiosis creates methane which causes stool to float.

4. Transit time under 36 hours

Measure transit with chlorophyllin: 1 tsp — time until bright green stool appears. Optimal: 18-24 hours. Acceptable: up to 36. Beyond 36: deconjugation becomes serious.

Note: Daily passage of stool does not guarantee passing transit time. A patient can have one bowel movement per day and still have 48+ hour transit.

Warning: Do not use chlorophyllin in copper-sensitive patients — chlorophyllin contains copper.

5. At least 2 fully formed stools per day.

Stool quantity is directly related to microbiome output.

6. Odor — faint, not foul

A healthy gut creates stool with minimal smell and CO₂ gas with no odor. A strong fecal or sulfurous odor points to a fermentation imbalance.

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If any test fails → Colon Protocol

All six tests must pass. Any failure indicates the colon environment needs restoration before other protocols can work effectively.

If all six pass → Small Intestine Protocol

The colon environment is already sound — uncommon, but possible. There is nothing to restore, so the Colon Protocol can be skipped; proceed to the Small Intestine Protocol.

Colon Protocol

pH is the master variable. Protocol entry point determined by stool pH reading.

Test stool pH

Use 6.0-8.0 pH paper strips, read at 15 seconds. Patient may prefer to wear gloves for any method.

Press pH paper against stool on toilet paper. This becomes harder as the microbiome heals and less stool remains on the paper. If stool smears on the pH paper, leave it in contact for 5 seconds, then wipe the smear off with fresh toilet paper. The color embedded in the paper reads clearly once the smear is cleared.

pH paper only registers on damp material. If the stool is dry on the surface, press the paper into the inside of the stool. In all cases, take the most alkaline reading. Do not test stool that has gone into the toilet water.

pH ≥8.0Pathologic dysbiosis
pH 7.6Severe dysbiosis
pH 7.2Moderate dysbiosis
pH 7.0Mild dysbiosis
pH 6.8Microbiome fragile
pH 6.4Microbiome acceptable
pH ≤6.0*Microbiome excellent
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pH ≥7.6 — Dysbiosis (extreme)

Urease-producing organisms (primarily Proteobacteria) are active. SCFA production is likely offline. The LAB seed bank may be empty.

Consider Acidulem: 4 per meal · Acetalac · Bacillimet: 3 per meal · Phylamet: 2 per meal. Zoiben: 1 tsp a day for 5 days (do not repeat) · Ellagica: 1 per meal

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pH 7.2-7.6 — Dysbiosis (high)

This is due to low SCFA output and/or urease-producing organisms (primarily Proteobacteria) dominant. The LAB seed bank may be empty.

Consider Acidulem: 6 per meal · Acetalac · Bacillimet: 4 per meal · Pokegeshi: 4 per meal · Phylamet: 4 per meal · Bifidus probiotics* · Zoiben: 1/2 tsp a day for 5 days (do not repeat) · Ellagica: 2 per meal · Clinical Electron Charger · Sextaphage* (Russian bacteriophage)

*Take on an empty stomach first thing in the morning, 10 minutes after 1/2 tsp sodium bicarbonate in a glass of water. The bicarbonate neutralizes stomach acid so it does not destroy the probiotics or bacteriophages before they reach the intestine.
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pH 6.8–7.2 — Dysbiosis (moderate)

Elevated pH is likely due to low SCFA production.

Consider Acidulem: 4 per meal · Bacillimet: 3 per meal · Phylamet: 2 per meal. Zoiben: 1/4 tsp a day for 5 days · Ellagica: 1 per meal

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pH 6.2–6.8 — Microbiome transition zone

SCFAs are being produced, but not yet at optimal levels.

Consider Acidulem: 2 per meal · Bacillimet: 2 per meal · Phylamet: 1 per meal · Panaceum: 4 per meal

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pH 5.7–6.2 — Microbiome quality restored

As long as the low pH is not due to carbohydrate malabsorption, the microbiome is fully producing SCFA. Congratulations.

Consider Acidulem: 1 per meal · Bacillimet 1 per meal · Panaceum: 2 per meal

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*pH ≤6.0 — Check for carbohydrate malabsorption

A pH at the acidic end of the strip reflects acid in the colon, and that acid can come from healthy SCFA production or from pathological fermentation of carbohydrate that was never absorbed upstream. The pH strip will not read below 6.0, so a true success reading and a malabsorption reading look identical on paper.

The deciding factor is context. In a patient who has not been on the protocol, an acidic reading is far more likely to be carbohydrate malabsorption than a pristine microbiome. Rule out carbohydrate malabsorption, short bowel syndrome, celiac disease, fructose intolerance, or lactose intolerance before treating the reading as a success.

Stool floats

Methane is being produced. Consider Allimet. Garlic extracts also lower propionate producers. Take the minimum dose for the minimum amount of time needed to achieve desired effect.

Stool consistency — a separate axis from pH

Hard to pass and infrequent

Drink more water and add organic psyllium powder mixed in water to the diet.

Mushy with pH 5.7–6.4

Something is irritating the stool. Look at spices, supplements, and drugs.

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Dietary window — for established dysbiosis not responding to supplementation

With elevated stool pH, consider lowering meat intake to 10% of the diet, and focusing on vegetables. For grains, consider flaked oats fermented with Lactobacillus Plantarum and millet.

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Either at the same time, or following, we begin the Small Intestine Protocol.

Small Intestine Protocol

Wants low microbial density — match the agent to the pathobiont. The two foundational columns come first; the three targeted agents follow. Ordered easy (top) to hard (bottom) to suppress.

Pathobiont (signature) Bacillus spores
(Bacillimet)		 Bile flow
(Glytamins)		 EO + bitters
(Zoiben)		 Bacteriophage
(Sextaphage)		 Thiosulfinate
(Allimet*)
Fermentative gram-negativesKlebsiella, E. coli — H₂, fermentation F F S S* S
Histamine / amine producersMorganella, Enterobacter — histamine, putrescine, cadaverine F F S P S*
Streptococcusfermentative gram-positive — lactate, D-lactate, H₂ F F S S* S
Staphylococcusopportunist gram-positive — toxins, inflammation F F S S* S
Proteolytic / putrefactiveProteus, Clostridia, Fusobacterium — ammonia, amines F F S* P S
Fungi / SIFOCandida albicans F F S* R S
Enterococcusbile-resistant gram-positive (VRE-type) — lactate, broad intrinsic resistance F F S S* S
SulfidogensBilophila wadsworthia, Desulfovibrio — H₂S F F S* R S
MethanogensMethanobrevibacter smithii — CH₄ F F P R S*
Key
F — foundational / restorative (Bacillimet and Bile): broad, non-eradicating action that restores and holds the compartment rather than targeting one organism — Bacillus spore pressure and biofilm disruption; bile’s detergent protection, peristalsis, and Bacillus germination.
S* — best in class: the lead targeted method for that pathobiont.
S — susceptible: the agent reaches and suppresses the organism.
P — partial: variable, strain-dependent, or indirect; lowers burden rather than eradicates.
R — resistant: the agent does not meaningfully act on the organism.
*Garlic extracts also lower propionate producers. Take the minimum dose for the minimum amount of time needed to achieve desired effect.
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A major part of small intestine restoration is improving bile flow — which takes us to the Gallbladder (Bile) protocol.

Gallbladder (Bile)

Bile is location-dependent: beneficial in intestines, caustic if retrograde

Diagnostics
Stool signsPale or clay-colored stool (bile not reaching intestine). Floating stool (can be fat malabsorption, but usually methane gas). Constipation unresponsive to fiber.
GB41 tendernessFirm pressure on Gallbladder 41 acupuncture point. Tenderness indicates gallbladder stress.
JaundiceYellowing of skin or sclera indicates bilirubin elevation from biliary obstruction.
Blood markersElevated bilirubin, ALP (alkaline phosphatase), GGT (gamma-glutamyl transferase). Elevated cholesterol may also indicate impaired bile excretion.
Symptoms after fatty foodsNausea, bloating, right upper quadrant pain or discomfort after meals with fat content.
UltrasoundUltrasound to confirm stones, sludge, wall thickening.
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Step 1 — Consider Glytamins, 1x per day for 10 days

If high chlorine exposure, add glycine and taurine.
Warning: Dry, cramping, or hot sensation in GB area → pause for a few days, resume at lower frequency.
If no resolution after 10 days ↓

Step 2 — Continue Glytamins + therapeutic ultrasound

Another 10 days with therapeutic ultrasound over gallbladder, 5 min/session. For liver stones, apply over liver.

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Optional — Traditional flush after 10 days

Standard olive oil / Epsom salt flush can be done after completing at least 10 days of the protocol, but is unnecessary. True stones sink — floating material is typically saponified oil (artifact).

Post-gallbladder-removal patients still need biliary support — stones can continue forming in liver and hepatic ducts.
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Consider a Glytamins course once a year, like an annual oil change. Now we move to the third microbiome — the sinuses.

Sinus & Brain Detox

Where the brain, immune system, lungs, and circulation converge

Step 1 — Detox stage

Bind fat-soluble toxins (mycotoxins, bacterial lipid toxins), dissolve biofilm matrix, recover mucin layer architecture, and support ciliary clearance. Add salt water sinus rinses to support clearance as needed.

Consider Ventrimet 6 sprays a day each nostril

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Step 2 — Postbiotic spray

Provide postbiotics to suppress mold and toxic bacteria, normalize immune response, and decrease neuro-inflammation. Run in parallel with Step 1.

Consider Sentrimet 6 sprays a day each nostril

After 2 weeks, or once sinuses are clear ↓

Step 3 — Sinus probiotics

  • Corynebacterium pseudodiphtheriticum
  • Dolosigranulum pigrum
  • Commensal Staphylococcus epidermidis

Consider Floramet 6 sprays a day each nostril

Raise the head of the bed 4-8 inches above the foot to support sinus and glymphatic drainage.
Nasal breathing matters. Mouth breathing bypasses the sinus entirely — no NO delivery to lungs or circulation.
Success markers
Morning TumescenceVascular/NO biomarker in both sexes. Penile erections in men; genital fullness/engorgement in women.
Increased lung capacityMeasurable improvement in breath hold time or spirometry as NO delivery to lungs improves.
Cognitive function improvesMemory, verbal fluency, focus, and processing speed improve as neuro-inflammation decreases.
Sinuses open upNasal airflow increases, congestion resolves, mouth breathing decreases.
Due to the labyrinthine nature of the sinuses, for optimum health, some clients may need to stay on the Sentrimet/Ventrimet protocol indefinitely.
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The sinuses are responsible for roughly 80% of the brain’s detox flow. Once it is detoxifying properly, the next part to assess is sleep quality — which takes us to the Sleep Apnea protocol.

Sleep Apnea

The condition we sleep through

Step 1 — Identify location(s)

SinusesVisual inspection: the anterior head of the inferior turbinates.
Experience: one or both nostrils become stuffy or blocked.
Protocol: Ventrimet, Sentrimet, and Floramet. Nasal strips can open the airway.
Soft palateVisual inspection: see the Mallampati classification system.
Experience: snoring.
Protocol: Ventrimet and Sentrimet sprayed into the back of the throat.
Tongue and epiglottisVisual inspection: a swollen tongue, possibly with scalloping on the edges.
Experience: airway closes when resting on one’s back.
Protocol: in addition to the sinus protocol, consider a Nightshift belt (3rd party, prescription) or a positional backpack (tennis-ball or wedge style). Warning: positional aids worsen sinus-driven apnea — where the sinuses are also involved, pair them with head-of-bed incline to keep the sinuses draining.
BrainVisual inspection: none.
Experience: apnea.
Protocol: not an airway blockage — the brain intermittently fails to send the signal to breathe, often tied to unstable CO₂/bicarbonate buffering and chronic hyperventilation. Work up acid-base status — see the Saliva & Urine pH protocol.
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Step 2 — Read the severity

Two ways to read the same story. The instrumented workup measures it; the dream and sleep signs cost nothing.

Instrumented — SpO₂ and/or heart rate monitoring during sleep:

  • Number of apneic events
  • Depth of desaturation (O₂ drop)
  • Length of desaturation
  • Elevated heart rate without desaturation indicates the apneic event was short enough not to desaturate

Free — dream and sleep signs:

  • Violent or suffocation dreams → apnea
  • Frustration dreams (running, stuck, searching) → hypopnea
  • Pounding heart or sweat-soaked sheets on waking → sympathetic surge from oxygen desaturation
  • Sheets and blankets in disarray → restless sleep from repeated arousal events
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Step 3 — Identify and address the inflammatory trigger

Mold/bacteria in home air and bedding, dust mites in bed, pollen season, pet allergies, allergens in diet, infection in sinuses/teeth/throat. See book for full differential.

The most common driver is the home itself — mold and bacteria in the air and bedding. Remediate it. See Home Detox tab.

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We are now almost ready to begin the heavy metal chelation protocol. But first, you must know how to measure your urine and saliva pH, which is explained in the next protocol.

Saliva & Urine pH

Two independent readings — saliva reads the bicarbonate system, urine reads the acid load

Step 1 — Morning saliva test

First thing on rising — before food, toothpaste, or any mouthwash. Rinse the mouth with water, then gently gnaw the tongue until saliva flows, and read the saliva pH at 15 seconds.

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Step 2 — Morning urine test

Read urine from mid stream flow at 15 seconds.

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Saliva — the bicarbonate system
8.0 — Excellent
7.6 — Good
7.2 — Fair
6.8 — Acceptable
6.4 — AcidicConsider Bicarbamet.
6.0 — Highly acidicConsider Bicarbamet.
A compromised bicarbonate system (saliva at the acidic end) warrants a workup: rule out chronic hyperventilation (lowers CO₂), dehydration, oral hygiene, and apnea / airway issues. Consider bicarbonates, chelation, and zinc. Check blood CO₂ if possible.
Poor salivary flow may read falsely acidic. Rule out stress, dehydration, and autoimmune disorders.

Consider Autonomis 1-2 caps a day during stressful moments

Urine — the acid load
7.2–8.0If not due to bicarbonate supplementation, consider a kidney workup.
6.4–6.8 — Excellent
6.0–6.4Consider Bicarbamet to buffer acidic dietary choices.
The strip floor hides the depth. The pH paper does not change color below 6.0, so a urine of 5.0 reads identical to 6.0 — ten times more acidic, same color. What the strip can’t show, the dose can: the more PM Bicarbamet needed to hold morning urine at 6.8, the more severe the acidosis.
If AM urine is ≤6.0 and/or saliva is ≤6.6 consider 2-6 Bicarbamet with 8 oz of water before sleep (so long as at least 4 hours have passed after the last meal) This may offset the negative effects that an overly acidic diet would otherwise create over time.
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Now that you know how to measure urine pH, we can begin heavy Metal Detox.

Metal Detox

Nature uses relay races, so should we

Step 1 — Identify metals

Hair, blood, urine, transdermal testing. See book for strengths and weaknesses of each method. Include mercury filling history (especially if removed improperly or gums turned dark) and history of MRIs with gadolinium contrast injection.

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Step 2 — Measure kidney function

Bloodwork (BUN, creatinine, eGFR) or urine test strips to determine detox capacity.

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Step 3 — Raise urine pH to 7.5 during chelation. Verify with pH paper.

Acidic urine can cause redistribution. For urine alkalinization, consider one Bicarbamet per 25 lb body weight and chelators in 8 oz of water on an empty stomach first thing in the morning, then wait 30 minutes before eating.

The relay — three tiers of chelator

Why a relay

Every tier used alone risks redistribution — solubilizing a metal in one place and dropping it in another. The relay hands the metal up an affinity gradient: a fast, small Tier 1 mobilizer reaches into crystalline deposits and tight binding pockets, passes its cargo to a more stable Tier 2, which passes it to a rigid Tier 3 terminal catcher that rarely lets go. Match the donor chemistry to the metal — hard donors for hard metals, soft donors for soft (HSAB; see remedylink.com/charts).

Hard-metal relay — good kidney function

  • Tier 1 — citrate or malate (malate where histamine is an issue): fast mobilizer; dissolves crystalline deposits and reaches small spaces
  • Tier 2 — EDTA (Medicardium V2O): stable mid-relay carrier
  • Tier 3 — DOTA (Captimet): macrocyclic terminal catcher
  • Add silica (methylsilanetriol) if aluminum is present

Soft-metal relay — good kidney function

  • Tier 1 — alpha-lipoic acid (ALA): fat-soluble soft-metal mobilizer; reaches lipid compartments
  • Tier 2 — MiaDMSA (Dasimet): stable mid-relay carrier
  • Tier 3 — DOTA (Captimet): the macrocyclic cage out-holds sulfur-donor chelators (Hg log K 26.4 vs DMSA 16.5), so DOTA is the terminal catcher for soft metals too
Arsenic is the exception: MiaDMSA (Dasimet) is the strongest available holder for arsenic and serves as its terminal (Tier 3) catcher.

Mixed burden — most patients

Most patients carry both chemistries. Run the hard relay and the soft relay together. DOTA (Captimet) is the shared Tier 3 terminal catcher for both sides — arsenic being the one exception, held by MiaDMSA.

How the protocol unfolds — build from the catcher up

Stage one tier at a time, by tolerance

Begin with the most stable runner and add the next mobilizer only once the current combination is shown to be tolerated, so anything a mobilizer frees always meets a more stable catcher already in place. Never mobilize more than you can capture.

1 — Tier 3 alone (DOTA / Captimet), low dose

The macrocyclic Tier 3 carries little redistribution risk — its stability keeps it from dropping cargo back into circulation, which makes it the safest single agent to start with. If 100 mg of a Tier 3 alone is tolerated, then go to 200 mg. If 200 mg is tolerated, you can add in Tier 2.

2 — Add Tier 2 once Tier 3 is tolerated

It is not about plateauing — it is about tolerance. Once Tier 3 is shown to be tolerated at the doses above, add the Tier 2 — EDTA for hard-metal coverage and/or MiaDMSA for soft. For sensitive patients, the molar dose of Tier 2 must always be lower than Tier 3.

3 — Add Tier 1 once Tiers 3 + 2 are tolerated together

Add Tier 1 if and only if Tiers 2 and 3 run at the same time are tolerated. Add citrate and malate for hard metals, alpha-lipoic acid for soft. Tier 1 reaches the small spaces the larger chelators cannot enter — protein binding pockets, crystal interstices, and enzyme active sites where the metal sits in the position of the missing essential cofactor. For sensitive patients, Tier 1 amounts must never exceed the molar dose of Tier 2 or Tier 3.

Adding Tier 1 is the riskiest move — it carries the greatest chance of redistribution. For patients with gadolinium symptoms, hold Tier 1 back for at least 6 months.
Urine pH: citrates may be enough to alkalinize the urine at this point, but test urine pH to be sure.

Standard Dosing schedule

1 Captimet, 1 Dasimet, 1 Medicardium V2O and 6 Bicarbamet 5 days on, 2 days off, with re-mineralizing on the off days — especially zinc, and magnesium and copper as needed.

Alkalinizing the sensitive patient: where Tier 1 citrates are contraindicated, alkalinization may have to be accomplished with sodium bicarbonate baths instead.
When the three-tier protocol plateaus ↓

Methyl DOTA (Vellumet) — intracellular reach

Fat-soluble macrocyclic; crosses cell membranes and demethylates to active DOTA inside the cell. This is the most aggressive level of the standard protocol, and the risk of mineral stripping is real. Bring magnesium and zinc to normal levels before starting, and replenish them on the off cycle (3–5 days on, 2 days off). Vellumet must always be taken with magnesium glycinate before, during, and after.

Run Vellumet for only 3 days, then follow with Captimet (DOTA). Always keep a gradient from the brain to the body — Vellumet frees metal from intracellular and brain compartments, and the Captimet that follows keeps it moving outward instead of redistributing back.

Weak kidney protocol

Sensitive / compromised kidney function consider:

  • Begin with 100mg DOTA (1 Captimet) a day 5 days on 2 days off for one month
  • If well tolerated, then try adding 100 mg EDTA (1 Medicardium V2O) for hard and borderline metals or 100 mg MiaDMSA (1 Dasimet) for soft metals
  • If both are needed, start with EDTA, then add MiaDMSA one week later

Capsules can be opened and absorbed sublingually if there are no metal fillings present in the mouth. Sensitive clients can start with 1/2 capsule.

Kidney stress warning: New lower back pain during chelation should be assumed to be stressed kidneys (barring physical injury). Stop protocol until discomfort subsides while taking CoQ10. Restart 1 week after last discomfort.
Serum CO₂ as progress marker: Metals break mitochondrial ATP production. As metals clear, ATP recovers → CO₂ rises on bloodwork. Below 27 suggests underperforming mitochondria and compromised Phase III.
Sequencing: Beginning a chemical detox without first restoring the microbiome and beginning metal chelation work is not suggested. See the book for further explanation.
Reading tubular damage from metals: Blood tests are preferred. Where they are unavailable, tenderness on firm pressure over the German (Voll) — not Chinese — acupuncture points Ki2b and Ki3 may indicate tubular damage from metals. See CHARTS for point locations.
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Now that the microbiome is restored and the metals are moving out, we can begin the Chemical Detox protocol.

Chemical Detox

Build in reverse order: Phase III before Phase II before Phase I

Step 1 — Identify ongoing exposures (including mold)

Remediate or avoid toxic exposure. No protocol overcomes continuous re-exposure.

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Step 2 — Baseline testing

Blood or urine test for toxicity baseline. If labwork is unavailable, firm pressure on acupuncture point LV3. Monitor for decreasing discomfort as protocol progresses.

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Step 3 — Complete Gallbladder protocol

Bile flow is the primary exit route for Phase II conjugated chemicals. Gallbladder (Bile) protocol precedes chemical detox.

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Step 4 — Lower stool pH to 6.5

See Colon Protocol. Dysbiotic gut bacteria produce beta-glucuronidase and sulfatases that strip Phase II conjugation tags, causing toxin reabsorption. Colon Protocol precedes chemical detox.

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Step 5 — Phase III support

Recover ATP necessary to power Phase III (ABC cassette pumps). Be at least 2 weeks into Metal Detox protocol before starting Chemical detox, or have blood CO₂ of 27.

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Step 6 — Phase II support

Supply conjugants: glutathione, glucuronolactone, sulfate, TMG, and B5.

In acute crisis, consider additional liposomal glutathione, glycine, alanine, glucuronolactone, and Calcium D-Glucarate.

Consider Xeneplex

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Step 7 — Phase I support

Oxidation (CYP450): fat-soluble toxin → reactive intermediate.

Consider Xeneplex

Phases must be supported in reverse order: III before II before I. Enhancing Phase I without Phase II = the intermediate is often MORE toxic than the parent compound. Enhancing Phase II without Phase III = conjugated toxins trapped inside cells. Never mobilize more than you can capture and drain.

Cyclodextrin binding — lipophilic toxins

HPBCD cyclodextrins encapsulate fat-soluble toxins, rendering them water-soluble for excretion — a parallel route to bile and oral binders for the lipophilic fraction.

Consider Albedextrin

Binders: Client may benefit from oral binders during active detox to catch conjugated toxins in the gut before bacterial beta-glucuronidase can deconjugate them. Psyllium (powder in water, not capsules), activated charcoal, or cholestyramine depending on situation. See book for more details.
Consider alternating Xeneplex, Glytamins and Albedextrin for 1 month, then reassess.
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Now we turn to one of the main toxins in our homes — mold.

Home Detox

If the building’s delivery rate of toxins exceeds the body’s clearance rate, no protocol will produce sustained improvement.

Why modern homes are vulnerable

OSB sheathing (broken cellulose surfaces). Paper-backed drywall. Cellulose insulation if borates leach. Roof penetrations (solar panels, skylights, vents). Vapor wrapping the home. Particle board furniture. All are pre-digested food for mold or moisture traps. Lignin in solid timber provides armor that processed wood products lack.

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Assessment tools

Moisture meterElevated readings where things should be dry — below windows, around plumbing, behind fixtures
Particle counterIndoor 10-micron counts >2× outdoor = mold growing inside (exclude cooking, smoking, talc, hair spray)
UV flashlightMany mycotoxins fluoresce — check pantries, refrigerators, food storage in low ambient light
Urine mycotoxin testingInformative when positive. False-negative risk: stored mycotoxins in fatty tissue may not show on unchallenged test.
Symptom patternBetter away from home. Worse in certain rooms. Seasonal fluctuation (humidity-dependent).
Sick, irritable, or forgetful house membersOther occupants showing cognitive, behavioral, or health changes — especially elderly.
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Remediation principles

Remove the water source first. Contaminated porous building materials should be removed, not cleaned. Non-porous surfaces can be treated. HEPA filtration during and after work.

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HVAC

Ductwork distributes contamination to every room. Skin shed (~2 lbs/year per occupant) feeds organisms in ducts.

Dehumidifier downstream of AC coilEliminates wet zone where mold grows inside the system
Blower upgrade for HEPA filtrationStock blowers cannot generate adequate flow through HEPA. Upgraded blower turns air handler into continuous cleaning system.
HVAC cleaning if ductwork is metalUse a cleaning service that vents dust outside the home.
Ductless mini-splits with through-wall ERVs eliminate ductwork entirely — the cleanest option.
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Fogging

Sanidate (peracetic acid + H₂O₂)Stronger antimicrobial. Sporicidal. For heavy remediation, attics, crawlspaces. Can affect electronics — cover or remove sensitive equipment. Breaks down to water, O₂, acetic acid.
Hypochlorous acid (HOCl)Same compound neutrophils use. Safer for electronics than Sanidate but still not risk-free — exercise caution. Can be generated at home from salt water (electrolysis). Sustainable, no ongoing supply cost. For maintenance fogging.

HEPA vacuuming of areas beforehand is highly recommended.

Consider our Home Fogging System

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Porous surfaces

Clothing: usually recoverable with multiple wash cycles using mycotoxin-specific enzyme formulations. Carpets and upholstery: typically not recoverable — plan on replacement. Hard surfaces clean readily. The line falls at porosity.

Minimum intervention if remediation is not possible: Turn off HVAC and open windows. Place high-quality air filter (HEPA + activated carbon) in bedroom at minimum. Wash all bedding, seal mattress in vapor barrier. Fog house monthly, bedroom every 2 weeks.
Gas stoves produce nitrogen oxides and ultrafine particulates with neurologic effects. Range hood venting to outside during cooking, or switch to electric.
Wood stoves must be used in positive pressure environment — combustion toxins can be pulled into living space through the chimney flue when not in use.
~25% of the population carries HLA-DR haplotypes predisposing to chronic mold-illness response. The unaffected housemate is not evidence that the affected one is imagining things.
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If you are still having health issues, we may need to address spike proteins.

Spike Protein Protocol

Never cleave without binding

Indication

History of complications after a COVID infection or COVID vaccination. Long COVID symptoms (fatigue, brain fog, cardiovascular symptoms, POTS).

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Diagnostics
D-dimerElevated D-dimer suggests ongoing spike protein–driven clotting activity.
Additional clotting markersFibrinogen, ESR (sed rate), PT/INR, platelet count.
Fingertip lancet (if labwork unavailable)Observe speed of drop formation, size of drop, time to stop bleeding. Abnormal clotting behavior is informative.
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Step 1 — Cleave: proteolytic enzymes

Liposomal proteolytic enzymes (serrapeptase, lumbrokinase, nattokinase). Liposomal delivery is required — non-liposomal enzymes will not reach the bloodstream. Cleaves intact spike protein.

Consider 6 Protelase 3x a day

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Critical warning

Cleaved spike protein breaks into approximately 7 prion fragments. Each fragment can template neighboring proteins into misfolded configurations. Proteolytic enzymes without cyclodextrin binding = iatrogenic prion exposure. Do not trade cardiovascular disease for neurodegeneration.

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Step 2 — Bind: cyclodextrins (simultaneous with Step 1)

HPBCD cyclodextrins encapsulate the prion fragments produced by cleavage, rendering them water-soluble for excretion. Steps 1 and 2 must run together — never cleave without binding.

Consider Albedextrin 1x a day

Both steps are simultaneous, not sequential. The binding must be present when the cleavage happens. Protelase and Albedextrin are used together.

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